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A middle-aged patient with arterial hypertension and dyslipidaemia presented with a 10-year history of recurrent sustained monomorphic ventricular tachycardia (VT) provoked by exertion or emotional affect (figure 1A). He had been treated with propafenone and amiodarone. Four years before, the standard 12-lead and modified Fontaine ECG in sinus rhythm were normal, as was the echocardiogram. VT of different morphology was reproducibly induced and terminated with programmed right ventricular (RV) pacing (figure 1B). Coronary angiography showed three-vessel coronary artery disease (figure 2A,B). The operator thought this …
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